García RMT

Idioma: Español
Referencias bibliográficas: 13
Paginas: 258-259
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FRAGMENTO

Las infecciones fúngicas en humanos son sumamente prevalentes en el ámbito mundial; afectan a ambos sexos, todas las edades, estratos socioeconómicos y zonas geográficas. Son causadas por levaduras (especies de Candida y Malassezia), dermatofitos (Microsporum, Epidermophyton y Trichophyton) y hongos no dermatofítos. La infección más común es por Trichophyton rubrum, que causa onicomicosis y tiñas.

REFERENCIAS (EN ESTE ARTÍCULO)

1. Vinh, D.C., “Insights into human antifungal immunity from primary immunodeficiencies”, The Lancet Infectious Diseases, 2011, 11: 780-792.

2. Serjeantson, S. y Lawrence, G., “Autosomal recessive inheritance of susceptibility to tinea imbricata”, The Lancet, 1977, 309: 13-15.

3. English, M., “Trichophyton rubrum infection in families”, Br Med J, 1957, 1: 744-746.

4. Zaias, N., Tosti, A., Rebell, G, et al., “Autosomal dominant pattern of distal subungual onychomycosis caused by Trichophyton rubrum”, J Am Acad Dermatol, 1996, 34.

5. Engelhardt, K. y Grimbacher, B., “Mendelian traits causing susceptibility to mucocutaneous fungal infections in human subjects”, J Allergy Clin Immunol, 2012, 129: 294-305.

6. Ferwerda, B., Ferwerda, G., Plantinga, T. et al., “Human Dectin-1 deficiency and mucocutaneous fungal infections”, N Engl J Med, 2009, 361: 1760-1767.

7. Glocker, E., Hennigs, A., Nabavi, M. et al., “A homozygous card9 mutation in a family with susceptibility to fungal infections”, N Engl J Med, 2009, 361: 1727-1735.

8. Zuccarello, D., Salpietro, D., Gangemi, S. et al., “Familial chronic nail candidiasis with icam-1 deficiency: a new form of chronic mucocutaneous candidiasis”, J Med Genet, 2002, 39: 671-675.

9. Jaradat, S.W., Cubillos, S., Krieg, N. et al., “Low defb4 copy number and high systemic hbd-2 and il-22 levels are associated with dermatophytosis”, J Invest Dermatol, 2015, 135 (3): 750-758.

10. Zaitz, C., Campbell, I. y Moraes, J., “hla-associated susceptibility to chronic onychomycosis in Brazilian Ashkenazi Jews”, Int J Dermatol, 1996, 35: 681-682.

11. García-Romero, M., Granados, J., Vega-Memije, M. et al., “Analysis of genetic polymorphism of the hla-b and hla-dr loci in patients with dermatophytic onychomycosis and in their first-degree relatives”, Actas Dermosifiliogr, 2012, 103: 59-62.

12. Kaya, T., Eskandari, G., Guvenc, U. et al., “cd4+cd25+ Treg cells in patients with toenail onychomycosis”, Arch Dermatol Res, 2009, 301: 725- 729.

13. Lanternier, F., Cypowyj, S., Picard, C. et al., “Primary immunodeficiencies underlying fungal infections”, Current Opinion in Pediatrics, 2013, 25: 736-747.