Rivero PR, Suárez BB, Benítez CY

Language: Spanish
References: 17
Page: 1-11
PDF size: 149.74 Kb.

ABSTRACT

Introduction: There is male over-mortality in nine of the ten leading causes of death in Cuba. The hegemonic model of masculinity, its essential attributes and the genetic characteristics that depend on this genre can constitute risk factors for health.
Objectives: To characterize the presence of indicators of hegemonic masculinity in men carriers of chronic diseases where it prevails over male mortality in Cuba, and to describe how the diseases that constitute the ten leading causes of death in Cuba can also be present in male relatives of 1st and 2nd degree of these cases.
Methods: A case study was conducted. An integrated analysis of the concept was carried out: hegemonic masculinity, expressed in a set of indicators related to this concept. Theoretical methods of investigation were used: the synthetic analytic, the transition from the abstract to the concrete, the inductive-deductive and the historicallogical. At the empirical level, the self administered questionnaire, the family tree and the individual interview in cases where it was necessary to deepen in the obtaining of data. 31 men from four provinces of Cuba, living in urban areas, were part of the sample.
Results: The analysis of cases, allowed to characterize the presence of nuclear aspects of the traditional way of being men that constitute risk factors for male health, and of relatives of 1st and 2nd degree of these men affected with diseases that are within the ten leading causes of death in Cuba.
Conclusions: refer to the need to incorporate the masculinities approach in the health care process of men at all levels, with emphasis on primary care. It was more frequent to find affected men of the ten diseases studied in families of the index cases, with diabetes mellitus, heart disease and chronic respiratory diseases being those with the greatest presence of affected male relatives.

REFERENCES

1. Salazar A. La desventaja masculina en salud. En: Rivero Pino R. Masculinidades y paternidades en Cuba. La Habana: Editorial CENESEX; 2015. p. 66-78.

2. Riuz M. Epigenética: Los hábitos del padre también dejan huellas en el hijo [en línea]. Barcelona: La Vanguardia. [actualizado 13 feb 2016]. Vida. Acceso: 20/12/2017. Disponible en: https://www.lavanguardia.com/vida/20160213/302119227627/epigenetica-habitospadre- dejan-huella-hijo.html

3. Donkin I, Soetkin V, Ingerslev LR, Quian K, Mechta K, Nordkap L et al. Obesity and Bariatric Surgery Drive Epigenetic Variation of Spermatozoa in Humans. Cell Metab [en línea]. 2016;23(2):369-78. Acceso: 26/12/2017. Disponible en: https://www.cell.com/cell-metabolism/fulltext/S1550-4131(15)00571-9

4. Rivero Pino R; Ulloa Guerra O. Reseñas de estudios cubanos sobre masculinidades. La Habana: Editorial CENESEX; 2017. p. 11-19.

5. Hernández de Armas Y. Impacto del modelo hegemónico de masculinidad en la salud cardiovascular masculina. Programa de superación para profesionales de enfermería. [Tesis Master en Sexología y Sociedad]. La Habana: Universidad de Ciencias Médicas de La Habana, Centro Nacional de Educación Sexual; 2018.

6. Rivero Pino R. Promoción-prevención local-comunitaria en salud genética. [Tesis de Maestría en Desarrollo Comunitario]. Santa Clara: Universidad Central "Martha Abreu" de Las Villas, Facultad de Ciencias Sociales; 2018.

7. Moran Gershoni, Shmuel Pietrokovski. The landscape of sex-differential transcriptome and its consequent selection in human adults. BMC Biol. 2017;15(1):7.

8. Viswanathan M, McCarthy MI, Snehalatha C, Hitman GA, Ramachandran A. Familial aggregation of type 2 (non‐insulin‐dependent) diabetes mellitus in South India; absence of excess maternal transmission. Diabet Med. 1996;13(3):232-7.

9. Wild S, Roglic G, Anders G, Sicree R, King H. Global prevalence of diabetes. Diabetes Care. 2004;27:1047-53.

10. Huxtable SJ, Saker PJ, Haddad L, Walker M, Frayling TM, Levy JC, Hitman GA, O'Rahilly S, Hattersley AT, McCarthy MI: Analysis of parent-offspring trios provides evidence for linkage and association between the insulin gene and type 2 diabetes mediated exclusively through paternally transmitted class III variable number tandem repeat alleles. Diabetes. 2000;49(1):126-30.

11. DeChiara TM, Efstradiatas A, Robertson EJ: A growth-deficiency phenotype in heterozygous mice carrying an insulin-like growth factor II gene disrupted by targeting. Nature. 1990;345(6270):78-80.

12. Lindsay RS, Dabelea D, Roumain J, Hanson RL, Bennett PH, Knowler WC: Type 2 diabetes and low birth weight: the role of paternal inheritance in the association of low birth weight and diabetes. Diabetes. 2000;49(3):445-9.

13. Tatiana K, Jan AS, Agnieszka O, Andrew R, Katarzyna S, Sofia M. Maternal and paternal influences on left ventricular mass of offspring. Hypertension. 2003;41(1):69- 74.

14. Chen C, Haolin Z, Yue Z, Rong L, Jie Q. Paternal history of diabetes mellitus and hypertension affects the prevalence and phenotype of PCOS. J Assist Reprod Genet. 2015;32(12):1731-9.

15. Howard DS, Min L, Michael Gaziano J, Kathryn MR, Robert JG, Julie EB. Maternal and paternal history of myocardial infarction and risk of cardiovascular disease in men and women. Circulation. 2001;104(4):393-8.

16. Idoia L, Jonatan RR, Francisco BO, Helle ML, Jaanus H, Toomas V. Intergenerational cardiovascular disease risk factors involve both maternal and paternal BMI. Diab Care. 2010;33(4):894-900.

17. Kinra S, Davey SG, Okasha M, McCarron P, McEwen J. Is maternal transmission of coronary heart disease risk stronger than paternal transmission? Heart. 2003;89(8):834-8.