Neurobiological mechanisms of nociception and antinociception

2004, Number S1

Rev Mex Anest 2004; 27 (S1)

Guevara U

Language: Spanish
References: 8
Page: 134-135
PDF size: 41.32 Kb.

Text Extraction

No abstract

REFERENCES

1— Coderre JT, Kats J, Vaccarino LA, Melsack R. Contribution of centralneuroplasticity to pathologycal pain: review of clinical and experimental evidence. Pain 1993;52:259-285.
2— Katz J, Kavanagh BP, Saudler AN, Nierenberg H, Boylan JF, Friedlander M, Shaw BF. Preemptive analgesia. Clinical evidence of neuroplasticity contributing to postoperative pain. Anesthesiology. 1992;77(3):439-46.
3— Woolf CJ, Thompson SWN. The induction and maintenance of central sensibilization is dependent on N-Methyl.D- Aspartic Acid receptor activation: implications for post-injury pain hypersensitivity states. Pain, 1991;44:293-299.
4— Iadorola MJ, Sanders SR, Draisci G. Neuropeptide and c-fos gene expression in periaqueductal grey (PAG) during peripheral inflammation, Pain. Suppl, 1990;5:S270.
5— Noguchi K, Dubner R, Ruda MA. Preproenkephalin mRNA in spinal dorsal horn neurons is induced by peripheral inflammation and is co-localized with Fos and Fos-related proteins. Neuroscience 1992;46:561-570.
6— Perkins F, Kehlet H. Chronic pain as an outcome of surgery. Anesthesiology 2000;93:1123.
7— Jia H, Xie YF, Xiao DQ, Tang JS. Involvement of GABAergic modulation of the nucleus submedius (SM) morphine induced antinociception. Pain 2004;108:28-35.
8— Seagrove LC, Susuky R, Dickenson AH. Electrophysiological characterizations of rat lamina I dorsal Horn neurons and the involvement of excitatory amino acid receptors. Pain 2004;108:76-78.