Revuelta PR, Díaz-Romero RM

Language: Spanish
References: 16
Page: 27-32
PDF size: 84.25 Kb.

ABSTRACT

Introduction: The window of infectivity described by Caufield in North-American children is a period during which children are more susceptible to the acquisition of Mutans streptococci (MS). As a consequence their dental caries susceptibility is increased. To date, this level of infectivity of MS has not been described in mexican children.
Objectives: To determine the level of infectivity of MS in a group of Mexican children and to identify the association between MS in saliva from mothers and their children. A second objective was to determine the relationship between the colony-forming units (CFU) of MS with the number of teeth in the children’s mouth and their saliva’s buffering capacity.
Materials and methods: A transversal, prospective and observational study was carried out on 71 healthy women who had their prenatal care at the INPer. At the time of the study their children were healthy and between 4-24 months of age. The children were grouped according to age: G1 comprised 35 children ‹12 month of age; G2 comprised 36 children ›12 months of age. Bacteriological studies of their saliva (mother and children) were performed using the Caries Risk Test (CRT) kit [Ivoclar, Vivadent].
Results: Exists direct relationship with the CFU of the mother (r = .356), as well as the number of decayed teeth of the mother (r = .350). The level of infectivity of MS in this group of children was on average 14.9 months.
Conclusions: There is a direct relationship between the CFU › 105 and with the number of erupted teeth in the children. The preventive programs should be initiated during pregnancy as an attempt to prevent the premature tranSMission of MS from mothers to their infants.

REFERENCES

1. Keyes PH. The infectious and tranSMisible nature of experimental dental caries. Arch Oral Biol 1960; 1: 304-20.

2. Caufield PW, Griffen AL. Dental caries. An infectious and tranSMissible disease 2000; 47-5: 1001-19.

3. Tanzer AM. Microbiology of dental caries: Slots J, Taubaman MA (eds.). Contenporary oral microbiology and inmunology. St Louis, Missouri: Mosby-Year Book, Inc; 1992, p. 420.

4. Brow LJ, Wall TP, Lazar V. Trends in total caries experience: permanent and primary teeth. J Am Dent Assoc 2000; 131: 223-31.

5. Caufield PW. Dental caries a traSMissible and infectious disease revisited: a position paper. Pediatric Dentistry 1997; 19(8): 491-8.

6. Newbrun E. Current regulations and recommendations concerning water fluoridation, fluoride supplements and topical fluoride agents. J Dent Res 1992; 71: 1255-65.

7. Russell MW, Childers Nk, Michalek SM. A caries vaccine? The state of the science of immunization against dental caries. Caries Res 2004; 28-3: 230-5.

8. Edelstein BL, Douglas CW. Dispelling the myth that 50 percente of US schoolchildren have never had cavity. Public Health 1995; 110: 522-30.

9. Caufield PW. Caries una enfermedad infecciosa y tranSMisible. Rev Academia Mexicana de Pediatría 2005; 17-1: 40-6

10. Wang R. Early chilhood caries. J AM Dent Assoc 2006: 137-2: 148-50.

11. Florio FM, Klein MI, Pererira AC. J Clin Pediatr Dent 2004; 28-4: 303-8.

12. Carletto KFP, Cornejo LS. Early acquisition of Streptococcus mutans for children. Acta Odontol Latinoam 2005; 18-2: 69-74.

13. Bericht HS. Evaluation caries screening test. J Dent Res 1985; 93: 204-12.

14. Oral Health Surveys. Basic methods. Third ed Ginebra, Suiza: WHO; 1987.

15. Feistein AR. Clinical Epidemiology. The architecture of clinical research. Philadelphia PA: WB Saunders Co. 1985.

16. Wiegand A, Stock A, Attin R. Impact of the acid flow rate on dentin erosion. J Dent 2006; 17.