medigraphic.com
Colegio de Medicos y Cirujanos República de Costa Rica

2009, Number 588

<< Back Next >>

Rev Med Cos Cen 2009; 66 (588)

Síndrome de brugada

Francis GM, Montero CLB

Language: Spanish
References: 18
Page: 207-211
PDF size: 404.90 Kb.


ABSTRACT

The Brugada Syndrome is a new clinical entity taking the attention of many clinical physicians. It had been related with sudden cardiac death and malignant arrhythmias especially in young population. This article presents a review of the most important aspects of this syndrome.


REFERENCES

  1. Antzelevitch C: Brugada syndrome. Pacing Clin Electrophysiol 29:1130-1159, 2006

  2. Antzelevitch C, Brugada P, Borggrefe M, et al: Brugada syndrome: report of the Second Consensus Conference: endorsed by the Heart Rhythm Society and the European Heart Rhythm Association. Circulation 111:659-670, 2005

  3. Brugada P, Brugada J. Right bundle branch block, persistent ST segment elevation and sudden cardiac death: a distinct clinical and electrocardiographic syndrome: a multicenter report. J Am Coll Cardiol. 1992;20: 1391–1396

  4. Benito B, Brugada R, Brugada J, Brugada P. Brugada Syndrome. Progress in Cardiovascular Diseases, Vol. 51, No. 1 (July/August), 2008: pp 1-22

  5. Brugada J, Brugada R, Antzelevitch C, et al: Longterm follow-up of individuals with the electrocardiographic pattern of right bundle-branch block and St segment elevation in precordial leads V1 to V3. Circulation 105:73-78, 2002

  6. Belhassen B, Glick A, Viskin S: Efficacy of quinidine in high-risk patients with Brugada syndrome. Circulation 110:1731-1737, 2004

  7. Brugada R, Brugada J, Antzelevitch C, et al: Sodium channel blockers identify risk for sudden death in patients with ST-segment elevation and right bundle branch block but structurally normal hearts. Circulation 101:510-515, 2000

  8. Brugada, P. Síndrome de Bugada. Vol. 12 Nº 3. Cardiovascular risk factors. 2003

  9. Casini S, Tan HL, Bhuiyan ZA, et al: Characterization of a novel SCN5A mutation associated with Brugada syndrome reveals involvement of DIIIS4-S5linker in slow inactivation. Cardiovasc Res 76: 418-429, 2007

  10. Di Diego JM, Cordeiro JM, Goodrow RJ, et al: Ionic and cellular basis for the predominance of the Brugada syndrome phenotype in males. Circulation 106: 2004-2011, 2002

  11. Eckardt L: Gender differences in Brugada syndrome. J Cardiovasc Electrophysiol 18:422-424, 2007

  12. Matsuo K, AkahoshiM, Seto S, et al: Disappearance of the Brugada-type electrocardiogram after surgical castration: a role for testosterone and an explanation for the male preponderance. Pacing Clin Electrophysiol 26:1551-1553, 2003

  13. Mok NS, Chan NY, Chiu AC: Successful use of quinidine in treatment of electrical storm in Brugada syndrome. Pacing Clin Electrophysiol 27:821-823, 2004 (6 Pt 1)

  14. Priori SG, Napolitano C, Gasparini M, et al: Natural history of Brugada syndrome: insights for risk stratification and management. Circulation 105:1342-1347, 2002

  15. Probst V, Denjoy I, MeregalIi PG, et al: Clinical aspects and prognosis of Brugada syndrome in children. Circulation 115:2042-2048, 2007

  16. Schulze-Bahr E, Eckardt L, Paul M, et al: Molecular genetics of the Brugada syndrome, in: Antzelevitch C, Brugada P (eds): The Brugada Syndrome: from Bench to Bedside (ed 1). Blackwell Publishing, 2005, p. 42-51

  17. Yan GX, Antzelevitch C. Cellular basis for the electrocardiographic J wave. Circulation. 1996;93:372–379.

  18. Yan GX, Antzelevitch C: Cellular basis for the Brugada syndrome and other mechanisms of arrhythmogenesis associated with ST-segment elevation. Circulation 100:1660-1666, 1999.




2020     |     www.medigraphic.com