Rodríguez NC, Méndez PJÁ, Patiño ZAM

Language: Spanish
References: 17
Page: 111-114
PDF size: 43.69 Kb.

ABSTRACT

Objective: Demonstrate the electrophysiological changes in postoperative gastric bypass patients secondary to vitamin B12 deficiency. Material and methods: We performed an observational, cross sectional, non-comparative, 6 postoperative gastric bypass patients. By performing electrophysiological studies, nerve conduction and myograph assessing each patient, and the determination of serum levels of vitamin B12. The results were analyzed with measures of central tendency mean and median. Results: After electrophysiological evaluation in patients postoperative gastric bypass, are sensory and motor nerve conduction in the normal range in 6 patients, the study myograph insertion activity showed normal resting activity with electrical silence, action potential of normal motor unit recruitment pattern completely. The serum vitamin B12 levels were within normal levels. Conclusion: The findings of the electrophysiological studies and vitamin B12 levels in the 6 postoperative gastric bypass patients were normal. This shows that after bypass surgery, follow up controlled by health professionals as well as a good patient adherence to medical treatment and nutritional control, may prevent the development of neuropathy due to vitamin deficiency B12.

REFERENCES

1. Esquide J, de Luis R, Valero C. Anestesia en la cirugía bariátrica. Revista de Cirugía Española 2004; 75(5): 273-9.

2. Rubio HMA. Manual de obesidad mórbida. Editorial Médica Panamericana. Madrid, España. 2006: 270.

3. Méndez PJA, Zamudio C. Tesis: Resultados perioperatorios del bypass gástrico abierto. UMAE 25, Monterrey Nuevo León. 2009.

4. Moreno E, Zugasti M. Cirugía bariátrica: Situación actual. Revista Médica Universidad Navarra Madrid, España 2004; 48(2): 66-71.

5. Cánovas B, Sastre J, Neblett A, López-Pardo R. Técnicas en cirugía bariátrica. Experiencia en 78 casos. Nutrición Hospitalaria 2006; 21(5): 567-72.

6. Rebolledo A, Basfi-fer K, Rojas P, Codoceo J, Inostroza J, Carrasco F, Ruz M. Evolutión and quality of the diet of women with severe and morbid obesity undergoing gastric bypass. Arch Latinoam Nutr. 2009; 59(1):7-13.

7. Forrellat M, Hernández P. Deficiencia de vitamina B12: ¿tratamiento oral o parenteral? Revista Cubana de Hematología, Inmunología y Medicina Transfusional 2009: 25(1).

8. Puri V, Chaudhry N, Goel S, Gulati P, Nehru R, Chowdhury D. Vitamin B12 deficiency: a clinical and electrophysiological profile. Electromyogr Clin Neurophysiol. 2005 Jul-Aug;45(5):273-84.

9. Méndez PJA, Almeraya J. Tesis: Tratamiento quirúrgico de la obesidad mórbida con bypass gástrico abierto experiencia de 12 años. UMAE 25, Monterrey Nuevo León. 2008.

10. Abarbanel J, Berginer V, Osimani A et al. Neurologic complications alter gastric restriction surgery for morbid obesity. Neurology 1987; 37: 196-200.

11. Thaisetthawatkul P, Collazo-Claveil M, Sarr G et al. A controlled study of peripheral neuropathy after bariatric surgery. Neurology 2004; 63: 1462-70.

12. Boyd K, Greenfield J, Imran A, Noor P. Neurologic complications after surgery for obesity. Muscle Nerve 2006; 33: 166-176.

13. Preston D. Electromyography and neuromuscular disorders. Editorial Elsevier Second edition. Estados Unidos, 2005.

14. DeLisa J, Hang J. Manual of nerve conduction velocity and clinical neurophysiology. Third edition. Ed. Rayen Press EUA 2007.

15. Pease W, Lew H, Johnson E. Johnson’s practical electromyography; 2007: 4th Ed. Editorial Lippincott Williams & Wilkins. 4th edition. EUA.

16. Martínez J, Civera M. Déficits nutricionales tras cirugía bariátrica. Servicio de Endocrinología y Nutrición. Hospital Clínico Universitario Valencia. Revista Española de Obesidad 2007; 5(1): 19-26.

17. Mauri S, Aguilar R, Ruiz B et al. Complicaciones nutricionales después del tratamiento quirúrgico de la obesidad: ¿Qué ocurre en el bypass gástrico? Endocrinología y Nutrición 2007; 54(4): 193-9.