2012, Number 1
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ABSTRACTObjective: To identify the herniosis (collagenosis) carrying patient, through the coexistence of other connective tissue pathologies, concomitantly with hernia, to be able to establish them, in the future, as predictive factors of recurrence and other complications; as well as to choose the appropriate surgery and establish recurrence probabilities and criteria for those other pathologies.
Setting: Specialized Center for the Treatment of Hernias, León, Gto. México.
Design: Prospective, longitudinal, and observational study.
Statistical analysis: Observational analysis expressed in percentages.
Material and methods: Forty-nine adults were operated for different types of hernia, searching for concurrent pathologies related to alterations of the connective tissue (collagenosis), familial antecedents of hernias, and smoking habit of the patient.
Results: Thirty-four (69%) patients had diseases related with herniosis (collagenosis). Diagnoses: hemorroids, 5; hiatal hernia, 5; varicose pelvic members, 5; colon diverticulosis, 4; vesicular litiasis, 3; epididymal cysts, 3; renal cysts, 2; ovarian cysts, 3; pulmonary emphysema, 3; hepatic polycystosis, 1; aortic aneurysm, 1; lipomatosis, 2; cutaneous grooves, 2; round ligament cyst, 1; joint laxity, 1. Three patients had four pathologies, seven had three, seven had two, and twenty had one. Positive for smoking were 28 (58%). Familial antecedents of hernias were found in 67%.
Conclusions: These findings establish a strong clinical correlation of hernia with other diseases related to herniosis, hence, they could be considered as predictive tools for recurrences and/or complications.
Keith A. On the origin and nature of hernia. Br J Surg 1924; 11: 455-475.
Read RC. Arthur Keith, the anatomist who envisioned herniosis. Hernia 2007; 11: 469-471.
Klinge U, Zheng H, Si ZY, Schumpelick V, Bhardwaj R, Klosterhalfen B. Synthesis of type I and III collagen, expression of fibronectin and matrix metalloproteinase 1 and - 13 in hernias of patients with inguinal hernia. Int J Surg Investing 1999; 1: 219-227.
Klinge U, Junge K, Mertens PR. Herniosis: a biological approach. Hernia 2004; 8: 300-301.
Cannon DJ, Read RC. Metastatic emphysema: a mechanism for acquiring inguinal herniation. Ann Surg 1981; 194: 270-278.
Hauer-Jensen M, Bursac Z, Read RC. Is herniosis the single etiology of Saint’s triad? Hernia 2009; 13: 29-34.
Drutz HP, Alarab M. Pelvic organ prolapse: demographics and future growth prospects. Int Urogynecol J Pelvic Floor Dysfunct 2006; 17: S6-S9.
Carley ME, Schaffer J. Urinary incontinence and pelvic organ prolapse in women with Marfan or Ehlers Danlos syndrome. Am J Obstet Gynecol 2000; 182: 1021-1023.
Al-Rawi ZS, Al-Rawi ZT. Joint hypermobility in women with genital prolapse. Lancet 1982; 319: 1439-1441.
Marshman D, Percy J, Fielding I, Delbridge L. Rectal prolapse: relationship with joint hypermobility. Aust NZ J Surg 1987; 57: 827-829.
Read RC. Signs of herniosis in women with vaginal prolapse and/or stress incontinence. Hernia 2008; 12: 449-452.
Amato G, Marasa L, Sciacchitano T, Bell SG, Romano G, Gioviale MC, et al. Histological findings of the internal inguinal ring in patients having indirect inguinal hernia. Hernia 2009; 13: 259-262.
Bump RC, McClish DK. Cigarette smoking and urinary incontinence in women. Am J Obstet Gynecol 1992; 167: 1213-1218.
Junge K, Rosch R, Anurov M, Titkova S, Ottinger A, Klinge U, et al. Modification of collagen formation using supplemented mesh materials. Hernia 2006; 10: 492-497.
Bendavid R. The unified theory of hernia formation. Hernia 2004; 8: 171-176.