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2003, Number 1

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Rev Acta Médica 2003; 11 (1)

Nutrition: an intervention strategy in the autistic child

Maciques RE
Full text How to cite this article

Language: Spanish
References: 16
Page:
PDF size: 46.99 Kb.


Key words:

autism, casein, gluten, nutrition, diet.

ABSTRACT

Autism is a generalized disorder of the child´s development. Starting early in the child’s life, autism is one of the most serious disorders of development, behaviour and communication, with neuropathological, neurophysiological, neurochemical and genetic foundations, among others. The autistic syndrome is a disorder especially refractory to both psycho-educational and biological treatments, thus making the autistic patient the subject of every kind of intervention, some of them without enough scientific support. Will be the gluten- and caseine-free diets one of them? Several lines of research can lead us to the role of the function of the opioid systems in the development of social behaviour. According to these theories, autism presents early in childhood because of an overload of the Central Nervous System with opioid peptides, affecting brain neurotransmitters. These peptides are probably of exogenous source, and derived in part from the incomplet digestion of gluten and casein. Favourable changes can be observed in those autistic children in whom certain gluten- and casein-containing foods has been removed from their diets, and the intake of sugars and some industrial products with chemical substances added has been reduced. In contrast with these observations, those autistics who have not sticked to such a diet, maintain their behavioural and comprehension disorders. What’s the bottomline? That there certain foods which might contain opoid peptides as intermediate products capable of reaching the brain and become active in cases of alterations of the intestinal permeability, thus leading some authors to proppose that hypendorphinic action might be regulated by means of dietetic restrictions. So, what is the solution? I think there is a challenge for professionals in the field of Nutrition and Neurosciences to find new perspectives and answers to the questions that still existing in this matter.


REFERENCES

  1. Sistema DSM-IV de Clasificación. Asociación Americana de Psiquiatría. 1993.

  2. Uta Frizt, 1991.

  3. Kanner, L. Los Trastornos autistas del contacto afectivo 1943:20.

  4. Kanner L. “Lenguaje irrelevante y metafórico en el autismo infantil precoz”, Leo Kanner, 1946, p.21

  5. Lenguaje irrelevante y metafórico en el autismo infantil precoz. 1946:20.

  6. Asperger H. La psicopatía autista en la niñez.1947:77.

  7. APA, 1994, pp.67-68.

  8. Lorna Wing, 1995.

  9. Edelson S. Visión Global del autismo. Centro para El estudio del Autismo. Salem, Oregón. 1995:4.

  10. Gilbert CH. Endogenus opioids and opiate antagonist in autism; brief review of empirical finding and implication for clinicians 1995; 35:239-45.

  11. Scifo R, Cioni M, Nicolasi, A. Opiod–inmune interactions in autism: behavioural and inmunologic asessment during a double – blind treatmente with Nalfrexone. 1995.

  12. Pavone L, Fiumara A, Bottaro G. Autism and celiac diseases. 1997; 42(1).

  13. Reichel KL. Biochemistry and Psychophysiology of autistic syndromes. 1994.

  14. Lucarelli S, Frediani T. Food allergy and infantile autism. 1995.

  15. Campell M. Treatment of autistic disorder. J Am Acad Child Adolesc Psychiatry 1996; 35(2): 13–143.

  16. Warren RP, Singh VK. Elevated serotonin levels in autism: association with the major histocompatibility complex. Neuropsychobiology 1996;34(2):72-5.




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Rev Acta Médica. 2003;11