Aguilera JT

Language: Spanish
References: 11
Page: 101-104
PDF size: 43.19 Kb.

ABSTRACT

Bronchiectasi is a syndrome that associates many etiologies. This term describes the abnormal, irreversible dilatation and thickening of the bronchi wall, causing inadequate clearance and pooling of mucus in airways, as well as inflammation and persistent microbial infection.

REFERENCES

1. Beirne PA, Banner NR, Khaghani A, et al. Lung transplantation on noncystic fibrosis bronchiectasis: Analysis of a 13 year experience, J Heart Lung transplant 2005: 24:1530.

2. Bilton D. Update on non-cystic fibrosis bronchiectasis. Curr Opin Pulm Med 2008; 14:595-599.

3. Elkins MR, Robinson M, Ross BR, et al. A control trial of lung-term inhaled hypertonic saline in patience with CF. N Engl J Med 2006; 354: 229-240.

4. Flume PA, Yankaskas JR, Eveling M, et al. Massive hemoptysis in CF. Chest 2005; 128:729-738.

5. Horvath I, Loukides S, Wodehouse T, et al. Increase levels of exhales carbon monoxide in bronchiectasis: A new marker of oxidative stress. Thorax 1998; 53:867-860

6. Keane MP, Arenberg DA,Moore BB, et al. CXC chemoqines and angiogenesis/angiostasis. Proc Assoc Am Physicians 1998;110:288- 296.

7. Nicotra MB, Rivera M, Dale AM, et al. Clinical, pathophysiologic, and microbiologic characterization of bronchectasis in an aging cohort. Chest 1995;108:955-961.

8. ODonnell AE. Bronchiectasis. Chest 2008;134:815-823

9. Prikk K, Maici P, Pirila E, et al. Invivo collagenase-2 (MM-8) expression by human bronchial epithelial cells and monocytes/ macrophages in bronchectasis. J Pathol 2001;194:232-238.

10. Rayner CF, Rutman A, Dewar A, et al. Ciliary disorientation in patients with chronic upper respiratory tract inflammation. Am J Respir Crit Care Med 1995;151:800-804.

11. Reid LM. Reduction in brochialsubdivision in bronchiectasis. Thorax 1995;5:233