Meaney E

Idioma: Inglés [English version]
Referencias bibliográficas: 11
Paginas: s160-163
Archivo PDF: 248.52 Kb.

REFERENCIAS (EN ESTE ARTÍCULO)

1. Ross R. Atherosclerosis- An inflammatory disease. N Engl J Med. 1999; 340: 115-126.

2. Libby P. Current concepts of the pathogenesis of the acute coronary syndromes. Circulation. 2001; 104: 365-372.

3. Gimbrone MA Jr, García-Cardeña G. Vascular endothelium, hemodynamics, and the pathobiology of atherosclerosis. Cardiovasc Pathol. 2013; 22: 9-15.

4. Wold D, Ley K. Immunity and inflammation in atherosclerosis. Circulation Res. 2019; 124: 315-327.

5. Moore K, Sheedy F, Fisher E. Macrophages in atherosclerosis: a dynamic balance. Nat Rev Immunol. 2013; 13: 709-721.

6. Bennett MR, Sinha S, Owens GK. Vascular smooth muscle cells in atherosclerosis. Circ Res. 2016; 118: 692-702.

7. Patti G, Melfi R, Di Sciascio G. The role of endothelial dysfunction in the pathogenesis and in clinical practice of atherosclerosis. Current evidences. Recenti Prog Med. 2005; 96: 499-507.

8. Watanabe N, Ikeda U. Matrix metalloproteinases and atherosclerosis. Curr Atheroscler Rep. 2004; 6: 112-120.

9. Heo K-S, Fujiwara K, Abe J-I. Shear stress and atherosclerosis. Mol Cells. 2014; 37: 435-440.

10. Nissen SE, Nicholls AJ, Sipahi I, Libby P, Raiclen JS, Ballantyne CM et al. Effect of very high-intensity statin therapy on regression of coronary atherosclerosis. The ASTEROID Trial. JAMA. 2006; 295: 1556-1565.

11. Bentzon JF, Otsuka F, Virmani R, Falk E. Mechanisms of plaque formation and rupture. Circ Res. 2014; 114: 1852-1866.