medigraphic.com
SPANISH

Revista Mexicana de Neurociencia

Academia Mexicana de Neurología, A.C.
  • Contents
  • View Archive
  • Information
    • General Information        
    • Directory
  • Publish
    • Instructions for authors        
  • medigraphic.com
    • Home
    • Journals index            
    • Register / Login
  • Mi perfil

2012, Number 1

<< Back Next >>

Rev Mex Neuroci 2012; 13 (1)

Cardiovascular evaluation of the autonomic function in syncope

González-Duarte A, Estañol-Vidal B, Oseguera-Moguel J
Full text How to cite this article

Language: Spanish
References: 9
Page: 30-35
PDF size: 270.77 Kb.


Key words:

Dysautonomy, orthostatic hypotension, postural orthostatic tachycardia syndrome, syncope, neurogenic syncope.

ABSTRACT

The presence of recurrent syncope, or a single syncope in a high-risk patient without evidence of structural heart disease, or even with a previously recognized heart condition that does not explain the event, are circumstances that require an evaluation of the autonomic nervous system in order to provide a precise diagnosis and to define a treatment plan. The goal of such evaluation is not to reproduce the syncope or associated symptoms, but to describe the type of autonomic response that generates the test. The stereotyped autonomic responses or hemodynamic patterns help to establish the syncope etiology. Of great importance is to investigate the presence of cardiologic abnormalities and, in case of their existence, it is necessary to refer the patient with a specialist as soon as possible. Orthostatic hypotension, postural orthostatic tachycardia syndrome (POTS) and neurogenic syncope are the three main groups recognized as syncope varieties. It is not the main goal of therapy to achieve normotension, but to improve postural symptoms, the time of orthostatic tolerance, and the performance of daily activities without syncope. Of vital importance is to avoid producing supine hypertension with treatment. Drug therapy is never adequate by itself and the strategy should vary depending on the orthostatic stress


REFERENCES

  1. Barón-Esquvias G, Martinez Rubio, A. Tilt Table test: State of the Art. Indian Pacing and Electrophysiology Journal 2003; 3: 239-52.

  2. Foster M. A text book of physiology. 5th. Ed. London: Macmillan; 1890.

  3. Medina E. Pruebas de Función Autonómica. Manual de Métodos Diagnósticos en Electrofisiología Cardiovascular 2006; Colombia: Ed. Panamericana; p. 131-40.

  4. Fitzpatrick AP, Theodorakis G, Vardas P, Sutton R. Methodology of head-up tilt testing in patients with unexplained syncope. J Am Coll Cardiol 1991; 17: 125-30.

  5. Stein KM, Slotwiner DJ, Mittal S, Scheiner M, Markowitz SM, Lerman BB. Formal analysis of the optimal duration of tilt testing for the diagnosis of neurally mediated syncope. Am Heart J 2001; 141: 282-8.

  6. Joyner M, Masuki S. Pots versus deconditioning: the same or different? Clin Auton Res 2008; 18: 300-7.

  7. Levine BD, Zuckerman JH, Pawelczyk JA. Cardiac atrophy after bed rest deconditioning: a nonneural mechanism for orthostatic intolerance. Circulation 1997; 96: 517-25.

  8. Winker R, Barth A, Bidmod D, et al. Endurance exercise training in orthostatic intolerance: a randomized, controlled trial. Hypertension 2005; 45: 391-8.

  9. Figueroa J, Basford J, Low, P. Preventing and treating orthostatic hypotension: as easy as A,B,C. Clev Clin J Med 2010; 77: 298-306.




2020     |     www.medigraphic.com

Mi perfil

C?MO CITAR (Vancouver)

Rev Mex Neuroci. 2012;13