Briones GJC, García OED, Zavala CJC, Campos CJC, Sandoval AOL, Briones VCG, Díaz de León PMA

Language: Spanish
References: 12
Page: 159-163
PDF size: 320.00 Kb.

ABSTRACT

Introduction: Acute pancreatitis during pregnancy ranges from one thousand to one in twelve thousand births, the most frequent cause is biliary disease, also associated with hypertriglyceridemia or pre-eclampsia and microangiopathic syndrome. Two phases are recognized in the pathogenesis: systemic inflammatory response, due to the release of mediator’s vessel active and toxic (first two weeks) and a second, in which occur the septic complications (third week onwards). The gallstones and alcoholism are 75 to 95% of the causes of acute pancreatitis, multiple organic failure and Ranson’s criteria, have a sensitivity of 88% to predict poor prognosis.
Objective: Is to present developments and the results of a series of cases of pancreatitis and pregnancy.
Material and methods: Number of cases, with fifteen patients clinical, biochemical and morphological admitted to intensive care with entry criteria.
Results: Has documented the etiology of biliary origin, were treated with medical and surgical management through laparoscopic cholecystectomy a case with CEPRE, follow-up of pregnancies until its resolution, a case concluded in incomplete abortion.
Discussion: Acute pancreatitis during pregnancy is a rare cause, but should be considered a serious complication, with a complex pathophysiology.
Findings: Acute pancreatitis can occur in three quarters, associated with biliary disease, 46% required laparoscopic cholecystectomy, and the basis of the diagnosis was clinical, biochemical and morphological.

REFERENCES

1. Sánchez LR, Chapa AO, Gutiérrez VR, Fernández HE. Utilidad del déficit de base como factor pronóstico en la pancreatitis aguda. Gac Méd Méx. 2003;139(2):108-111.

2. Vázquez RJG. Pancreatitis aguda y embarazo. Rev Asoc Mex Med Crit y Ter Int. 1997;11(4):117-120.

3. Briones GJC, Castañón GJA, Díaz de León PM, Briones VCG. La unidad de cuidados intensivos multidisciplinaria y la medicina crítica en gineco-obstetricia. Rev Asoc Med Crit y Ter Int. 1996;10(6):276-279.

4. Díaz de León PM, Galeano MTM, García RD, Briones GJC, Gómez-Bravo TE. Diagnóstico y tratamiento de la pancreatitis aguda grave (PAG). Rev Asoc Med Crit y Ter Int. 2003;17(3):104-110.

5. Arenas OJ, Salazar ED, Martínez GH, Vadillo BM, Briones GC, Gómez CJG. Efecto de la infusión intraduodenal de lidocaína sobre pancreatitis aguda experimental. Cir Ciruj. 2008;76:473-479.

6. Sánchez LR, Acosta RAV, Chapa AO, Hurtado LLM. Etiología como determinante de severidad en la pancreatitis aguda. Gac Méd Méx. 2003;139(1):27-31.

7. Sánchez LR, Camacho HMI, Vega ChRG, Garza FJH, Campos CC, Gutiérrez VR. Pancreatitis aguda: experiencia de cinco años en el Hospital General de México. Gac Méd Méx. 2005;141(2):123-127.

8. Sánchez LR, Chapa AO, Vega ChRG, Garza FJH, Campos CC, Gutiérrez VR. Frecuencia de falla orgánica en la pancreatitis aguda necrótica. Gac Méd Méx. 2005;141(3):175-179.

9. Trejo LJA, Contreras GN, Barrera RJM, Ayala GF, Sánchez PLA. Síndrome de HELLP relacionado con pancreatitis aguda. Med Int Mex. 2006;22:465-468.

10. Torres CJ, vadillo BM, González BD. Pancreatitis hipertrigliceridémica durante el embarazo. Comunicación de un caso. Ginecol Obstet Mex. 2006;74:170-176.

11. Gonzalo I, Berzosa J, Díaz M. Pancreatitis aguda en el embarazo. Clin Invest Gin Obst. 2008;35(1):29-31.

12. Herrera GM, Chávez TN, Lizardi CJ. Pancreatitis aguda y embarazo. Médica Sur, México. 2003;10(2):101-104.