García-González AC, Orozco-Olguín P, Félix-Bullman J, Rodríguez-Weber F, Díaz-Greene E

Language: Spanish
References: 0
Page: 337-342
PDF size: 503.40 Kb.

ABSTRACT

Etiology of pericardial disease is varied and complex, pericardium responds in most cases not very specific, usually limited to inflammation of the layers and increased production of pericardial fluid. This paper reports the case of a 73-year-old female patient, with hypertension and hypothyroidism who began her current condition a week previous her entering with upper respiratory tract infection that remitted within two days without treatment. Subsequently, patient had dyspnea with moderate exercise progressing to small efforts dyspnea accompanied by nonproductive cough, so patient assisted to assessment, where vital signs were within normal limits, slight pallor of integuments and decreased heart sounds. EKG showed complexes with minimally alternating voltage; thorax radiography reported cardiomegaly grade I-II due to global growth, bilateral pleural effusion of left predominance and echocardiography with pericardial effusion generalized estimated between 400-500 cc, besides diastolic collapse of the right ventricle and systolic collapse of right atrial with significant variations in the rates of transmitral and transtricuspid flow; respiratory cycle echocardiogram reported pericardial effusion of about 1,000 cc with data of hemodynamic impairment. TAC confirmed pericarditis. It was decided to perform a pericardial window and drainage of pericardial effusion obtaining 700 cc of dark hematic pericardial effusion; stains, biopsy, antibodies were sent without finding the cause of pericardial effusion.