medigraphic.com
SPANISH

Anales de Radiología, México

  • Contents
  • View Archive
  • Information
    • General Information        
    • Directory
  • Publish
    • Instructions for authors        
  • medigraphic.com
    • Home
    • Journals index            
    • Register / Login
  • Mi perfil

2015, Number 3

<< Back Next >>

Anales de Radiología México 2015; 14 (3)

Bilateral calcinosis in striated, pale, and dentate nuclei and hypoparathyroidism: discussion of a case

Sucre-Grimaldo FM, Casares-Cruz K, Sandoval-Paredes J
Full text How to cite this article

Language: Spanish
References: 12
Page: 285-291
PDF size: 1222.62 Kb.


Key words:

Fahr’s disease, Fahr’s syndrome, bilateral calcification of basal ganglia, calcinosis, dementia, brain disease.

ABSTRACT

Fahr’s disease was described by Karl Theodor Fahr, in 1930, as idiopathic or hereditary calcifications of the basal nuclei associated with both neurological and psychiatric abnormalities. We present the case of a patient with secundary calcifications in basal nuclei identified by means of image studies.


REFERENCES

  1. Mufaddel AA, Al-Hassani GA. Familial idiopathic basal ganglia calcification (Fahr`s disease). Neurosciences 2014;19(3):171-7.

  2. Saleem et al. Fahr’s syndrome: literature review of current evidence. Orphanet Journal of Rare Diseases 2013;8:156.

  3. Ahad MA, Bala C, Karim S. Fahr’s syndrome. Bangladesh Medical Journal Khulna. 2013;45(1–2):33-35.

  4. Abubakar SA, Saidu S. Idiopathic bilateral strio-pallidodentate calcinosis (Fahr’s disease): A case report and review of the literature. Ann Afr Med 2012;11:234-7.

  5. Asokan AG, D’souza S, Jeganathan J, Pai S. Fahr’s Syndrome- An Interesting Case Presentation. J Clin Diagn Res. 2013;7(3):532-3.

  6. Hegde A, Mohan S, Lath N et al. Differential Diagnosis for Bilateral Abnormalities of the Basal Ganglia and Thalamus. RadioGraphics 2011;31:5-30.

  7. Shouyama M, Kitabata Y, Kaku T, Shinosaki K. Evaluation of Regional Cerebral Blood Flow in Fahr Disease with Schizophrenia-Like Psychosis: A Case Report. AJNR Am J Neuroradiol 2005;26:2527-2529.

  8. Da Silva RJ, Pereira IC, Oliveira JR. Analysis of Gene Expression Pattern and Neuroanatomical Correlates for SLC20A2 (PiT-2) Shows a Molecular Network with Potential Impact in Idiopathic Basal Ganglia Calcification (“Fahr’s Disease”). J of Molec Neuro MN 2013;50(2):280-283.

  9. Juha´ sz C., Haacke E., Hu J. et al. Multimodality Imaging of Cortical and White Matter Abnormalities in Sturge-Weber Syndrome AJNR Am J Neuroradiol 28:900-06.

  10. Moore D., Ye F., Schiffmann R., Butman J. Increased Signal Intensity in the Pulvinar on T1-Weighted Images: A Pathognomonic MR Imaging Sign of Fabry Disease. AJNR Am J Neuroradiol 24:1096-110.

  11. Manyam BV, Walters AS, Naria KR. Bilateral striopallidodentate calcinosis: Clinical characteristics of patients seen in a registry. Mov Disord 2001;16:258-6.

  12. Manyam BV. What is and what is not ‘Fahr’s disease’ Parkinsonism Relat Disord. 2005;11(2):73-80.




2020     |     www.medigraphic.com

Mi perfil

C?MO CITAR (Vancouver)

Anales de Radiología México. 2015;14