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Colegio de Medicina Interna de México.
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2005, Number 6

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Med Int Mex 2005; 21 (6)

Spontaneous thrombosis of jugular vein. A report of three cases and literature review

García FRLF, Mas MAP
Full text How to cite this article

Language: Spanish
References: 8
Page: 471-480
PDF size: 46.24 Kb.


Key words:

thrombosis, jugular vein, causes, manifestations, treatment.

ABSTRACT

Feminine patient of 56 years old that initiates its present suffering three days previous to its medical valuation with occipital migraine of sharp type, intermittent, accompanied by vertigo to the shifts of position. In addition, she presented pain in the right ear with irradiation to the ipsilateral shoulder, with partial improvement to conventional analgesics. Studies of image that corroborate the presence of the internal jugular venous thrombosis, with negative profile of hypercoagulable state were made; they were repeated three months after the event, which is reported again without alterations. The thrombosis of the jugular vein refers to the presence of a clot that occurs anywhere, from the origin of the jugular vein below the skull to the point in where the vein is united with the subclavian vein. The most frequent clinical manifestations are: leukocytosis, fever, cervical pain and mass in neck. The deficiency of protein C, S or antithrombin III predisposes the patient to a spontaneous intravascular thrombosis; nevertheless, a familiar history or antecedent of arterial thrombosis exists. The Doppler ultrasound can be useful to detect changes in the flow, secondary to the presence of a clot during the acute phase of its formation. The computed axial tomography is considered the study of election in case of thrombosis suspicion of the internal jugular vein. Once the diagnosis is made the use of anticoagulant therapy should be considered. The internal jugular venous thrombosis constitutes an uncommon cause of venous thrombosis. Its more frequent cause is the use of central venous catheter; nevertheless, in the cases of spontaneous thrombosis it will be necessary to rule out the hypercoagulable state and the malignant disease. More studies are required to establish the guides of treatment in these cases of thromboembolic disease.


REFERENCES

  1. García-Frade LF, Hurtado R. Alteraciones hematológicas en el embarazo. En: Manual de diagnóstico para el consultorio. 1a ed. México: Litoral, 2004.

  2. Dacey M. Internal jugular thrombosis. Kent County Hospital.

  3. Vázquez F. Bilateral internal jugular thrombosis associated with thrombophilia after ovarian induction for infertility. Medicina (B Aires) 2002;62(4):328-30.

  4. Cushman M. Estrogen plus progestin and risk of venous thrombosis. JAMA 2004;292(13):1573-80.

  5. Mammen E. Oral contraceptive pills and hormonal replacement therapy and thromboembolic disease. Hematol Oncol Clin North Am 2000;14(5):1045-59.

  6. Moores L, Bilello K, Murin S. Sex and gender issues and venous thromboembolism. Clin Chest Med 2004;25:281-97.

  7. Humphries K, Gill S. Risks and benefits of hormone replacement therapy: the evidence speaks. CMAJ 2003;168(8): 1001-10.

  8. Whiteman T, Hassouna H. Hypercoagulable states. Hematol Oncol Clin North Am 2000;14(2):355-77.




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C?MO CITAR (Vancouver)

Med Int Mex. 2005;21